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Therapeutic to Block Toxic Protein Spread in Neurodegeneration

Value Proposition· Disease-Modifying Mechanism: Elevates fibrinogen-like protein 1 (FGL1) in the brain to inhibit proteinopathies (spread of toxic proteins in the brain).· Versatile Delivery Options: Flexible treatment strategies including gene therapy, exosome delivery, and blood exchange to deliver FGL1.· Dual application: Functions...

Published: 1/22/2026 | Inventor(s): Xiaobo Mao, Ted Dawson, Valina Dawson, Wenjing Wang, Junkai Hu, Mingyao Ying, Jun Wang

Keywords(s):

Category(s): Clinical and Disease Specializations > Neurology > Parkinson’s Disease, Clinical and Disease Specializations > Neurology > Parkinson's Disease, Clinical and Disease Specializations > Neurology > Neurodegeneration, Clinical and Disease Specializations > Neurology > Alzheimer's Disease

Disulfide Bond-free and Fibril-specific Nanobodies Prevent the Prion-like α-synuclein Spreading

Unmet NeedLewy bodies dementia (LBD) is one of the most common causes of dementia, including Parkinson's disease with dementia (PDD) and dementia with Lewy bodies (DLB). Both clinical and experimental observations support the hypothesis of prion-like α-syn driving α-synucleinopathy, hence targeting the prion-like α-syn may be a new approach to treat...

Published: 1/22/2026 | Inventor(s): Xiaobo Mao, Wenjing Wang, Yemima Raini, Yuqing Liu, Ramhari Kumbhar

Keywords(s):

Category(s): Technology Classifications > Therapeutic Modalities > Therapeutic Antibodies, Clinical and Disease Specializations > Neurology > Alzheimer's Disease, Clinical and Disease Specializations > Neurology > Dementia, Clinical and Disease Specializations > Neurology > Parkinson’s Disease